SHORT NILEVAR DESCRIPTION:
Nilevar is an oral derivate of nortestosterone (nandrolone). The activity of this steroid is that of a mild to moderate anabolic, which is accompanied by an equally distinguishable androgenic and estrogenic component. The typical dose is 30-50 mg per day.
Nilevar only partially usefull in bodybuilding, it would only be of some use if it was the only anabolic steroid available at location. Otherwise it is a waste of time as it has the same or even worse side effects than Dianabol while its anabolic effect is mush smaller.
LONG NILEVAR DESCRIPTION: Nilevar is a trade name for the oral steroid norethandrolone, developed by the pharmaceutical firm Searle in the mid 1950’s. Norethandrolone is a derivative of nortestosterone (nandrolone), containing an added c17 alpha ethyl group so the molecule can withstand oral administration. The activity of this steroid is that of a mild to moderate anabolic, which is accompanied by an equally distinguishable androgenic and estrogenic component. This item was the predecessor to Anavar (oxandrolone), which was introduced to the US drug market about a decade later. When comparing the quality and overall effect of the two substances, oxandrolone is considered to be notably superior to the more androgenic and estrogenic norethandrolone. This is probably why we do not see this steroid much anymore. It has been off U.S. shelves for many years now, and is currently marketed only in Australia, France and Switzerland.
Although structurally nandrolone and norethandrolone are very similar, they seem to behave quite differently in the body. For starters, androgenic side effects such as oily skin, acne and body/facial hair growth seem to be slightly more pronounced with this drug. In this regard Nilevar is usually thought to more closely resemble a steroid such as Dianabol than a mild anabolic like Deca. And since the 5-alpha reduced metabolite of norethandrolone is weaker than its parent (just as we see with nandrolone), Proscar would offer us no benefit in reducing such symptoms (this drug is really only applicable with testosterone compounds). One might even run the risk of developing (or aggravating) a male pattern hair loss condition with Nilevar, although this steroid is admittedly still much less potent here than many stronger androgens including testosterone and Anadrol 50. Those with a familial predisposition for baldness would still be much better served with an injectable nandrolone preparation such as Deca-Durabolin, which is in all respects a better steroid.
And while we see a very low tendency for estrogen conversion with nandrolone, unfortunately again norethandrolone seems to have a reputation as a much more estrogenic steroid. This is likely due to the fact that it converts to a more biologically active form of estradiol, 17alpha ethyl estradiol, instead of regular estrogen. A similar problem is noted with Dianabol, which is much more troublesome than its non-alkylated cousin boldenone. Excessive water retention is the usual result with norethandrolone, producing an unsightly smoothness and loss of definition to the physique. In addition, the extra estrogen can also lead to the development of gynecomastia very quickly. It may therefore be advisable to include an anti estrogen such as Nolvadex and/or Proviron from the start of a cycle, in order to keep these side effects to a minimum. We also have the option of using the antiaromatase Arimidex, which is a much more effective remedy. It will efficiently stop the compound from converting to estrogen, essentially halting related side effects. The high selling price for this product makes it quite costly to use however, especially when we are taking it to treat the effects of what is considered to be a cheap and crude oral steroid.
Norethandrolone will also suppress endogenous testosterone production quite readily with use. This may be a result of not only its androgenic and estrogenic activity, but its action as a progestin as well. As mentioned when discussing Deca-Durabolin, 19-norandrogens are often shown to exhibit some affinity for the progesterone receptor. This tendency seems to be notably heightened in Nilevar, and contributes not only to its ability to suppress testosterone production, but also its propensity to induce fat storage and gynecomastia (side effects normally associated with estrogen). In order to help restore hormonal balance after each cycle a combination of HCG and Clomid/Nolvadex may prove very useful. While HCG is not always indicated with many of the milder anabolics, the strong suppressive nature of norethandrolone makes having this drug on-hand almost a necessity (especially for those inclined to notice testicular atrophy during steroid intake).
Clearly Nilevar cannot be looked at as an oral alternative to Deca-Durabolin, as this compound is much more troublesome. The increased tendency for estrogen conversion (when possible) and lowered anabolic effectiveness that results when a steroid is 17-alpha alkylated make norethandrolone very inefficient for building muscle. In administering an effective amount of steroid in terms of muscle growth, the user has to deal with much more in terms of side effects compared to nandrolone. The accumulation with norethandrolone is also going to be bloated mass, and not the quality muscularity we associate with Deca. A notably more pronounced strength increase may result with this substance, not doubt partly due to the extra fluid retention. The androgenic component also makes this steroid a less than ideal choice for women, who would be better served by an injectable nandrolone such as Durabolin.
Those who find an interest to take Nilevar most commonly find a daily dosage of 30-40 mg (3-4 tablets) is needed to receive an anabolic effect. In order to keep blood levels more constant, the tablets are also taken in divided doses (spread evenly throughout the day). As mentioned, norethandrolone is a c-17 alpha alkylated steroid in order to make oral dosing possible. Although a methyl group is typically used for this purpose with anabolic/androgenic steroids, in this case the steroid carries an ethyl substitution (just as we see with Orabolin). This is just as stressful to the liver of course, so the length of each cycle best kept to a minimum to avoid any serious damage. A logical duration would probably be no longer than 6 to 8 weeks, after which a longer break (from all related oral compounds) should be taken.
Although this steroid is sometimes circulated in Europe, it is not commonly found in the U.S. The demand for it is quite low, so large volume steroid dealers are not very interested in importing it. The only exception as of late seems to be the Jurox item Anaplex from Australia. This has been readily exported from Australia to Mexico, and thereafter has been reaching the States. As a result many athletes have been experimenting with this steroid recently, but admittedly it still does remain relatively unpopular on the black market. Although Anaplex is quite inexpensive compared to the older French Nilevar, one should not be tempted to use excessive dosages for a stronger anabolic effect. The level of estrogenic side effects and strain placed on the liver are certainly to be compounded as the dosages go up, making such a practice less than worthwhile in terms of gain vs. side effects. Dianabol is clearly a better alternative as far as orals go, and as already mentioned Deca-Durabolin is a much more tolerable option in terms of a nandrolone.